机译:肌肉特异性激酶重症肌无力IgG4自身抗体导致小鼠严重的神经肌肉接头功能障碍
1 Department of Human Genetics, Medical Genetics Centre, Leiden University Medical Centre, Leiden, The Netherlands 2 Department of Neurology, Leiden University Medical Centre, Leiden, The Netherlands 3 Department of MCB-Neurophysiology, Leiden University Medical Centre, Leiden, The Netherlands 4 Department of Neuroscience, School of Mental Health and Neuroscience, Faculty of Health, Medicine and Life Sciences, Maastricht University, Maastricht, The Netherlands 5 BAC BV, Naarden, The Netherlands 6 Department of Paediatric Neurology, Canisius Wilhelmina Hospital, Nijmegen, The Netherlands 7 Neuroimmunology Group, Biomedical Research Institute (BIOMED), Hasselt University, Diepenbeek, Belgium;
机译:肌肉特异性激酶重症肌无力IgG4自身抗体导致小鼠严重的神经肌肉接头功能障碍
机译:IgG4对肌肉特异性激酶的自身抗体经过Fab-Arm交换在肌炎肌无力患者中
机译:患者自身抗体耗尽了突触后肌肉特异性激酶,导致小鼠ACh受体支架和重症肌无力的拆卸。
机译:麝香Myasthenia的致病性IgG4亚类自身抗体
机译:肌肉特异性激酶(MUSK)引起的重症肌无力的小鼠模型,直接蛋白相互作用调节大电导(BK)钙激活钾通道的门控和转运
机译:患者自身抗体耗尽了突触后肌肉特异性激酶导致小鼠ACh受体支架和重症肌无力的拆卸
机译:肌肉特异性激酶myasthenia gravis IgG4自身抗体导致小鼠的严重神经肌肉结功能障碍