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机译:塞来昔布通过抑制胰岛素样信号传导延长环氧线虫的寿命,但不抑制环氧合酶2的活性
Department of Internal Medicine Division of Geriatric Medicine;
Department of Molecular and Integrative Physiology University of Michigan Medical School Ann Arbor MI 48109 USA;
Division of Medicinal Chemistry College of Pharmacy The Ohio State University Columbus OH USA;
3′-phosphoinositide-dependent kinase-1; Caenorhabditis elegans; celecoxib; cyclooxygenase-2 inhibitor; insulin-like signaling; longevity; nonsteroidal anti-inflammatory drug;
机译:塞来昔布通过抑制胰岛素样信号传导延长环氧线虫的寿命,但不抑制环氧合酶2的活性
机译:塞来昔布通过抑制胰岛素样信号传导延长环线虫的寿命,但不抑制环氧合酶2的活性
机译:哒嗪通过激活NRF2 / SKN-1信号通路诱导抗逆性并延长线虫寿命
机译:INS-12的生理功能,其中一种γ胰岛素样肽,C.秀丽隐杆线
机译:自噬基因atg-18通过神经肽信号传导非自主地调节秀丽隐杆线虫的寿命细胞。
机译:Celecoxib通过抑制胰岛素样信号传导但不含环氧化酶-2活性而延伸C. elegans寿命。
机译:塞来昔布通过抑制胰岛素样信号传导而不是环氧合酶-2活性来延长秀丽隐杆线虫的寿命