Interneuron Transplantation Rescues Social Behavior Deficits without Restoring Wild-Type Physiology in a Mouse Model of Autism with Excessive Synaptic Inhibition

摘要

Author contributions: D.G.S., D.V., J.L.R., and V.S.S. designed research; D.G.S., H.S., R.M., and K.L. performed research; D.G.S., R.M., and V.S.S. analyzed data; D.G.S. and V.S.S. wrote the paper.