机译:Saposin C刺激生长和侵袭,激活MAPK的p42 / 44和SAPK / JNK信号通路并上调前列腺癌和基质细胞中的uPA / uPAR表达
Department of Microbiology, Immunology and Parasitology,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Stanley S. Scott Cancer Center,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Stanley S. Scott Cancer Center,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Department of Medicine,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Department of Neurosciences, University of California, San Diego, Center for Molecular Genetics, La Jolla, California 92093, USA;
Department of Microbiology, Immunology and Parasitology,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Stanley S. Scott Cancer Center,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Department of Urology, Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Kekulé-Institut fuer Organische Chemie Und Biochemie, Universitaet Bonn, D-53121 Bonn, Germany;
Kekulé-Institut fuer Organische Chemie Und Biochemie, Universitaet Bonn, D-53121 Bonn, Germany;
Department of Microbiology, Immunology and Parasitology,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Stanley S. Scott Cancer Center,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Department of Microbiology, Immunology and Parasitology,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
Stanley S. Scott Cancer Center,Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA;
saposin C; prostate cancer; uPA/uPAR; prosaposin; invasion; growth factor; SAPK/JNK; MAPK; MMP; c-Jun;