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Insights into Fracture Repair Using a Murine Model of Osteofibrous Dysplasia

机译：使用骨纤维发育不良的小鼠模型洞察骨折修复

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作者
Xie, Wei Xiang.;
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作者单位

University of Toronto (Canada).;

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授予单位 University of Toronto (Canada).;
学科
学位 M.Sc.
年度 2019
页码 142 p.
总页数 142
原文格式 PDF
正文语种 eng
中图分类 AAI22621046;
关键词
fracture repair; osteofibrous dysplasia;

机译：骨折修复;骨纤维发育不良;

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1. Neonatal osteofibrous dysplasia associated with pathological tibia fracture: A case report and review of the literature [J] . ?etinkayaM., ?zkanH., K?ksalN., Journal of pediatric orthopaedics. Part B . 2012,第2期

机译：新生儿胫骨纤维异常增生伴病理性胫骨骨折1例并文献复习
2. Overview of Biological Mechanisms and Applications of Three Murine Models of Bone Repair: Closed Fracture with Intramedullary Fixation, Distraction Osteogenesis, and Marrow Ablation by Reaming [J] . Beth Bragdon, Kyle Lybrand, Louis Gerstenfeld Current Protocols in Mouse Biology . 2015,第1期

机译：三种小鼠骨修复模型的生物学机制及其应用概述：髓内固定闭合骨折，牵张成骨和扩孔骨髓切除术
3. TGFβ Inhibition Stimulates Collagen Maturation to Enhance Bone Repair and Fracture Resistance in a Murine Myeloma Model [J] . Green Alanna C, Lath Darren, Hudson Katie, Journal of bone and mineral research: the official journal of the American Society for Bone and Mineral Research . 2019,第12期

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4. Insight into Hydraulic Fracture Geometries Using Fracture Modeling Honoring Field Data Measurements and Post-Fracture Production [C] . Neil Stegent, Ron Dusterhoft, Hari Menon EAGE Conference and Exhibition . 2019

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5. Development and Characterization of Selective CB2R Inverse Agonists as a Novel Anti-inflammatory Therapy for Neuroinflammation: Mechanisms of Action in Murine and Human Microglia Models Provide Valuable Insights into Their Therapeutic Potential [D] . Alghamdi, Sahar Saleh. 2020

机译：选择性CB2R逆激动剂作为一种新型抗炎疗法的开发和特征，成为神经肾性炎症的抗炎治疗：小鼠和人微胶质模型的作用机制为其治疗潜力提供了有价值的见解
6. The Haploinsufficient Hematopoietic Microenvironment Is Critical to the Pathological Fracture Repair in Murine Models of Neurofibromatosis Type 1 [O] . Xiaohua Wu, Shi Chen, Yongzheng He, 2011

机译：该Haploinsufficient造血微环境是至关重要的神经纤维瘤病类型的小鼠模型的病理性骨折修复1
7. The Haploinsufficient Hematopoietic Microenvironment Is Critical to the Pathological Fracture Repair in Murine Models of Neurofibromatosis Type 1 [O] . Wu, Xiaohua, Chen, Shi, He, Yongzheng, 2011

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Insights into Fracture Repair Using a Murine Model of Osteofibrous Dysplasia

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