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Effect of the tight junction modulator Zonula occludens toxin (Zot) and the P-glycoprotein inhibitor itraconazole on brain transport enhancement and their potential for renal drug interactions

机译：紧密连接调节剂Zonula咬合毒素（Zot）和P-糖蛋白抑制剂伊曲康唑对脑转运增强的作用及其与肾脏药物相互作用的潜力

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作者
Karyekar, Chetan S.;
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作者单位

University of Maryland Baltimore;

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授予单位 University of Maryland Baltimore;
学科
学位 Ph.D.
年度 2002
页码 259 p.
总页数 259
原文格式 PDF
正文语种 eng
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外文文献

1. Enhancing the permeation of marker compounds and enaminone anticonvulsants across Caco-2 monolayers by modulating tight junctions using zonula occludens toxin. [J] . Cox DS, Gao H, Raje S, European journal of pharmaceutics and biopharmaceutics: official journal of Arbeitsgemeinschaft fuer Pharmazeutische Verfahrenstechnik e.V . 2001,第2期

机译：通过使用小带闭塞毒素调节紧密连接，增强标记化合物和烯胺酮抗惊厥药在Caco-2单层上的渗透。
2. Affinity purification and partial characterization of the zonulin/zonula occludens toxin (Zot) receptor from human brain. [J] . Lu R, Wang W, Uzzau S, Journal of Neurochemistry: Offical Journal of the International Society for Neurochemistry . 2000,第1期

机译：从人脑对zonulin / zonula咬合毒素（Zot）受体进行亲和纯化和部分表征。
3. Tight junction modulation and biochemical characterisation of the zonula occludens toxin C-and N-termini. [J] . Schmidt E, Kelly SM, van-der-Walle CF FEBS letters. . 2007,第16期

机译：小带的紧密连接调节和生化特征封闭了毒素C-和N-末端。
4. Enhanced oral delivery of hydrophilic drugs: Design and evaluation of tight junction modulators and cyclic peptide prodrugs. [D] . Ouyang, Hui. 2002

机译：增强亲水性药物的口服递送：设计和评估紧密连接调节剂和环肽前药。
5. Zonula occludens toxin modulates tight junctions through protein kinase C-dependent actin reorganization in vitro. [O] . A Fasano, C Fiorentini, G Donelli, 1995

机译：Zonula闭塞毒素通过蛋白激酶C依赖的肌动蛋白重组在体外调节紧密连接。
6. Zonula occludens toxin modulates tight junctions through protein kinase C-dependent actin reorganization, in vitro. [O] . Fasano, A, Fiorentini, C, Donelli, G, 1995

机译：Zonula闭塞毒素通过蛋白激酶C依赖的肌动蛋白重组在体外调节紧密连接。

Effect of the tight junction modulator Zonula occludens toxin (Zot) and the P-glycoprotein inhibitor itraconazole on brain transport enhancement and their potential for renal drug interactions

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