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Aberrant Acetylation of Mitochondrial Proteins, due to Loss of SIRT3, Results in Tumor Permissive Phenotypes and Increases Breast Cancer Malignancy Risk.

机译：线粒体蛋白的乙酰化异常，由于SIRT3的丢失，导致肿瘤允许表型的出现，并增加了乳腺癌的恶性风险。

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作者
Zou, Xianghui.;
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作者单位

Northwestern University.;

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授予单位 Northwestern University.;
学科 Biology.
学位 Ph.D.
年度 2017
页码 148 p.
总页数 148
原文格式 PDF
正文语种 eng
中图分类
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专利

1. Manganese Superoxide Dismutase Acetylation and Dysregulation, Due to Loss of SIRT3 Activity, Promote a Luminal B-Like Breast Carcinogenic-Permissive Phenotype [J] . Zou Xianghui, Santa-Maria Cesar Augusto, OBrien Joseph, Antioxidants and redox signalling . 2016,第6期

机译：锰超氧化物歧化酶乙酰化和失调，由于SIRT3活性的丧失，促进发光B样乳腺癌致癌性表型。
2. Manganese Superoxide Dismutase Acetylation and Dysregulation, Due to Loss of SIRT3 Activity, Promote a Luminal B-Like Breast Carcinogenic-Permissive Phenotype [J] . Zou Xianghui, Santa-Maria Cesar Augusto, OBrien Joseph, Antioxidants and redox signalling . 2016,第6期

机译：锰超氧化物歧化酶乙酰化和呼吸剂量，由于SIRT3活性的损失，促进了腔B样乳腺癌致癌杂散表型
3. SIRT3 is a mitochondrial tumor suppressor: A scientific tale that connects aberrant cellular ROS, the Warburg effect, and carcinogenesis [J] . HaigisM.C., DengC.-X., FinleyL.W.S., Cancer research: The official organ of the American Association for Cancer Research, Inc . 2012,第10期

机译：SIRT3是线粒体肿瘤抑制剂：将异常的细胞ROS，Warburg效应和致癌作用联系起来的科学故事
4. Bio-molecular optimized interactions and conformational switches in human ERβ and SRC-1 protein: An in silico approach to suppress tumor in breast cancer malignancies: Computational insight for suppression of tumorigenesis in breast malignancies [C] . Arundhati Banerjee, Sujay Ray 2016 International Conference on Microelectronics, Computing and Communications . 2016

机译：人类ERβ和SRC-1蛋白中生物分子的优化相互作用和构象转换：抑制乳腺癌恶性肿瘤的计算机方法：抑制乳腺癌恶性肿瘤发生的计算机洞察
5. The effect of cellular and acellular components of the tumor microenvironment on both tumor and stromal cell phenotypes in a breast cancer model system [D] . McLane, Joshua 2015

机译：乳腺癌模型系统中肿瘤微环境的细胞和非细胞成分对肿瘤和基质细胞表型的影响
6. Manganese Superoxide Dismutase Acetylation and Dysregulation Due to Loss of SIRT3 Activity Promote a Luminal B-Like Breast Carcinogenic-Permissive Phenotype [O] . Xianghui Zou, Cesar Augusto Santa-Maria, Joseph OBrien, -1

机译：锰超氧化物歧化酶乙酰化和失调由于SIRT3活性的损失促进发光B类乳腺癌致癌性表型。
7. SIRT3 and SIRT4 are mitochondrial tumor suppressor proteins that connect mitochondrial metabolism and carcinogenesis [O] . 2014

机译：SIRT3和SIRT4是连接线粒体代谢和致癌作用的线粒体肿瘤抑制蛋白
8. SIRT3 is a Mitochondrial Tumor Suppressor and Genetic Loss Results in a Murine Model for ER/PR-Positive Mammary Tumors Connecting Metabolism and Carcinogenesis SIRT3 is a Mitochondrial Tumor Suppressor. [R] . Gius, D. 2012

机译：sIRT3是线粒体肿瘤抑制因子和遗传丢失结果，用于ER / pR阳性乳腺肿瘤的小鼠模型连接代谢和癌变sIRT3是线粒体肿瘤抑制因子。

Aberrant Acetylation of Mitochondrial Proteins, due to Loss of SIRT3, Results in Tumor Permissive Phenotypes and Increases Breast Cancer Malignancy Risk.

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