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Exocyst Dysfunction Leads to Defects in Urothelial Differention in a Novel Mouse Model of Congenital Ureter Obstructions.

机译：囊外功能障碍导致先天性输尿管梗阻的新型小鼠模型中尿道分化的缺陷。

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作者
Lee, Amanda.;
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作者单位

University of Hawai'i at Manoa.;

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授予单位 University of Hawai'i at Manoa.;
学科 Cellular biology.;Developmental biology.;Molecular biology.
学位 Ph.D.
年度 2017
页码 107 p.
总页数 107
原文格式 PDF
正文语种 eng
中图分类
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入库时间 2022-08-17 11:54:08

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1. Fibroproliferative response to urothelial failure obliterates the ureter lumen in a mouse model of prenatal congenital obstructive nephropathy [J] . Amanda J. Lee, Noemi Polgar, Josephine A. Napoli, Scientific reports. . 2016,第1期

机译：对尿液衰竭的纤维增生反应丧失了产前先天性梗阻性肾病的小鼠模型中的输尿管内腔
2. Adamts18 deletion results in distinct developmental defects and provides a model for congenital disorders of lens, lung, and female reproductive tract development Adamts18 deletion results in distinct developmental defects and provides a model for congenital disorders of lens, lung, and female reproductive tract development Adamts18 deletion results in distinct developmental defects and provides a model for congenital disorders of lens, lung, and female reproductive tract development [J] . Alessandra Piersigilli, Sarah E. Earp, Yakir Guri, Biology Open . 2016,第11期

机译：Adamts18缺失会导致明显的发育缺陷，并为晶状体，肺和女性生殖道发育的先天性疾病提供模型。Adamts18缺失会导致明显的发育缺陷，并为晶状体，肺和女性生殖道发育的先天性疾病提供模型。导致明显的发育缺陷，并为晶状体，肺和女性生殖道发育的先天性疾病提供了模型
3. Adamts18 deletion results in distinct developmental defects and provides a model for congenital disorders of lens, lung, and female reproductive tract development Adamts18 deletion results in distinct developmental defects and provides a model for congenital disorders of lens, lung, and female reproductive tract development Adamts18 deletion results in distinct developmental defects and provides a model for congenital disorders of lens, lung, and female reproductive tract development [J] . Alessandra Piersigilli, Sarah E. Earp, Yakir Guri, Biology Open . 2016,第11期

机译：Adamts18缺失会导致明显的发育缺陷，并为晶状体，肺和女性生殖道发育的先天性疾病提供模型。Adamts18缺失会导致明显的发育缺陷，并为晶状体，肺和女性生殖道发育的先天性疾病提供模型。导致明显的发育缺陷，并为晶状体，肺和女性生殖道发育的先天性疾病提供了模型
4. Analysis of Retinal Degeneration in a Leber Congenital Amaurosis Mouse Model Using High Spatial Resolution MALDI-Imaging Mass Spectrometry [C] . David M. G. Anderson, Zsolt Ablonczy, Yiannis Koutalos, ASMS Conference on Mass Spectrometry and Allied Topics . 2016

机译：利用高空间分辨率MATDI成像质谱法分析LEBER先天性生物疲劳小鼠模型的视网膜变性
5. Oxidative Stress and Proteomic Studies of Mammalian Models of Age-Related Metabolic Dysfunction in Neurodegenerative Disorders: (1) Upregulation of Pin1 in Alzheimer Mouse Model (2) Analysis of Brain Post-Intranasal Administration of Insulin in Alzheimer and Induced Diabetic Mouse Models (3) Analysis Of Proteome Changes In The Parietal Lobule Of Rhesus Monkey Non-Human Primate Following Resveratrol Supplementation as a Part of a Western-Diet. [D] . Swomley, Aaron Michael. 2017

机译：氧化应激和蛋白质组学研究的神经退行性疾病的年龄相关代谢功能障碍的哺乳动物模型：（1）阿尔茨海默病小鼠模型中Pin1的上调（2）鼻内注射胰岛素对老年性痴呆症小鼠和诱导的糖尿病小鼠模型的分析（3）白藜芦醇补充作为西方饮食的一部分，恒河猴非人类灵长类动物顶叶小叶蛋白质组变化的分析。
6. Fibroproliferative response to urothelial failure obliterates the ureter lumen in a mouse model of prenatal congenital obstructive nephropathy [O] . Amanda J. Lee, Noemi Polgar, Josephine A. Napoli, -1

机译：对尿道上皮衰竭的纤维增生反应消除了产前先天性阻塞性肾病小鼠模型中的输尿管腔
7. Urothelial Defects from Targeted Inactivation of Exocyst Sec10 in Mice Cause Ureteropelvic Junction Obstructions. [O] . Ben Fogelgren, Noemi Polgar, Vanessa H Lui, 2015

机译：小鼠内皮细胞sec10靶向失活引起的尿路上皮缺损导致输尿管肾盂连接障碍。

Exocyst Dysfunction Leads to Defects in Urothelial Differention in a Novel Mouse Model of Congenital Ureter Obstructions.

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