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机译:通过重塑肿瘤微环境促进vorinostat和辛伐他汀的可变形脂质体促进抗肿瘤反应
State Key Laboratory of Drug Research Shanghai Institute of Materia Medica Chinese Academy of Sciences 501 Haike Rd Shanghai 201203 China;
State Key Laboratory of Drug Research Shanghai Institute of Materia Medica Chinese Academy of Sciences 501 Haike Rd Shanghai 201203 China;
State Key Laboratory of Drug Research Shanghai Institute of Materia Medica Chinese Academy of Sciences 501 Haike Rd Shanghai 201203 China;
State Key Laboratory of Drug Research Shanghai Institute of Materia Medica Chinese Academy of Sciences 501 Haike Rd Shanghai 201203 China;
State Key Laboratory of Drug Research Shanghai Institute of Materia Medica Chinese Academy of Sciences 501 Haike Rd Shanghai 201203 China;
State Key Laboratory of Drug Research Shanghai Institute of Materia Medica Chinese Academy of Sciences 501 Haike Rd Shanghai 201203 China;
State Key Laboratory of Drug Research Shanghai Institute of Materia Medica Chinese Academy of Sciences 501 Haike Rd Shanghai 201203 China;
State Key Laboratory of Drug Research Shanghai Institute of Materia Medica Chinese Academy of Sciences 501 Haike Rd Shanghai 201203 China;
State Key Laboratory of Drug Research Shanghai Institute of Materia Medica Chinese Academy of Sciences 501 Haike Rd Shanghai 201203 China;
机译:利用多针对脂质体乳蛋白酶重塑胶质瘤治疗的改造肿瘤免疫微环境(时间)
机译:缺氧 - / HIF-1α驱动因子肿瘤微环境阻碍抗肿瘤免疫应答和促进恶性进展
机译:IL-36γ改变肿瘤微环境并促进1型淋巴细胞介导的抗肿瘤免疫反应。
机译:姜黄素的新型抗肿瘤机制:改变肿瘤代谢的含义,重构肿瘤微环境和增强髓鞘
机译:免疫检查点B7X促进肿瘤微环境中调节性T细胞的诱导,并介导对免疫疗法的抗性
机译:PNAS Plus:IL-15是促进抗肿瘤反应的肿瘤微环境中炎症环境的组成部分
机译:利用多针对脂质体乳蛋白酶重塑胶质瘤治疗的改造肿瘤免疫微环境(时间)
机译:基质Caveolin-1的缺失是否通过激活src介导的pEaK1和pI3K途径重塑肿瘤微环境。