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机译:致病性分枝杆菌的巨噬细胞入侵机制
Department of Internal Medicine, Washington University School of Medicine, St. Louis, MO 63110, USA.;
Amino Acid Sequence; Animals; Complement 2 physiology; Complement 3 metabolism; Complement 3 Convertase metabolism; Complement 3b immunology; Horses; Humans; In Vitro; Isoflurophate pharmacology; Macrophages immunology microbiology; Mice; Molecular Sequence Data; Mycobacterium pathogenicity; Mycobacterium avium Complex immunology pathogenicity; Mycobacterium bovis immunology pathogenicity; Mycobacterium leprae immunology pathogenicity; Mycobacterium tuberculosis immunology pathogenicity; Opsonins; Research Support; Non-U.S. Gov't; Research Support; U.S. Gov't; P.H.S.; Virulence;
机译:维生素D和视黄酸的协同作用限制了致病性分枝杆菌对巨噬细胞的侵袭。
机译:分枝杆菌涉及Cd43胞外域的巨噬细胞入侵机制。
机译:致病性和非致病性分枝杆菌对巨噬细胞蛋白激酶C同工型的差异调节
机译:深度定量蛋白质组学,揭示治理分枝杆菌碳代谢的调节机制。
机译:表征原发性巨噬细胞对致病性和非致病性分枝杆菌感染的差异反应。
机译:分枝杆菌涉及Cd43的胞外域的巨噬细胞入侵机制。
机译:分枝杆菌涉及Cd43的胞外域的巨噬细胞入侵机制。