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机译:小鼠修补突变体中神经细胞命运和髓母细胞瘤的改变
Department of Developmental Biology, Stanford University School of Medicine, Stanford, CA 94305-5427, USA.;
Abnormalities; Multiple genetics; Animals; Body Patterning; Cell Lineage; Central Nervous System cytology embryology; Cerebellar Neoplasms genetics pathology; Ectoderm metabolism; Endoderm metabolism; Gene Expression Regulation; Developmental; Genes; Tumor Suppressor; Heterozygote; Homozygote; Medulloblastoma genetics pathology; Membrane Proteins genetics physiology; Mesoderm metabolism; Mice; Mice; Inbred C57BL; Mutation; Oncogene Proteins genetics; Research Support; Non-U.S. Gov't; Transcription Factors genetics;
机译:N-myc改变成神经母细胞瘤小鼠模型中肿瘤前细胞的命运
机译:N-myc改变成神经母细胞瘤小鼠模型中肿瘤前细胞的命运
机译:N-myc改变了髓母细胞瘤小鼠模型中肿瘤前细胞的命运。
机译:辐照后添加维生素C会降低高LET碳离子辐照的A_L细胞的突变率并改变CD59〜-突变的光谱
机译:神经发育过程中的不对称EGFR分布以及神经干细胞的默认细胞命运。
机译:N-myc改变成神经母细胞瘤小鼠模型中肿瘤前细胞的命运
机译:N-myc改变成神经母细胞瘤小鼠模型中肿瘤前细胞的命运