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机译:自闭症相关的SHANK3单倍剂量不足会导致人类神经元的I-h通道病变
Stanford Univ, Dept Mol & Cellular Physiol, Sch Med, 265 Campus Dr, Stanford, CA 94305 USA;
Stanford Univ, Dept Mol & Cellular Physiol, Sch Med, 265 Campus Dr, Stanford, CA 94305 USA|Stanford Univ, Inst Stem Cell Biol & Regenerat Med, Sch Med, 265 Campus Dr, Stanford, CA 94305 USA|Stanford Univ, Sch Med, Dept Pathol, 265 Campus Dr, Stanford, CA 94305 USA;
Stanford Univ, Dept Mol & Cellular Physiol, Sch Med, 265 Campus Dr, Stanford, CA 94305 USA;
Stanford Univ, Dept Mol & Cellular Physiol, Sch Med, 265 Campus Dr, Stanford, CA 94305 USA;
Stanford Univ, Dept Mol & Cellular Physiol, Sch Med, 265 Campus Dr, Stanford, CA 94305 USA;
Stanford Univ, Inst Stem Cell Biol & Regenerat Med, Sch Med, 265 Campus Dr, Stanford, CA 94305 USA|Stanford Univ, Sch Med, Dept Pathol, 265 Campus Dr, Stanford, CA 94305 USA;
Stanford Univ, Dept Mol & Cellular Physiol, Sch Med, 265 Campus Dr, Stanford, CA 94305 USA|Stanford Univ, Howard Hughes Med Inst, Sch Med, 265 Campus Dr, Stanford, CA 94305 USA;
机译:自闭症相关的Shank3基因的单倍剂量不足会导致突触功能,社交互动和社交沟通方面的缺陷
机译:单分子荧光原位杂交揭示人类SHANK3 mRNA表达在发育过程中和自闭症相关的SHANK3杂合性中发生变化
机译:人类多能干细胞来源的皮质神经元在自闭症中的高通量药物筛查:SHANK3单倍剂量不足综合征的概念研究证明。
机译:SCN3 A基因的启动子分析和钠通道β1亚基启动子活性的改变:对人类通道病的影响
机译:人髓磷脂碱性蛋白基因的单倍剂量不足和中枢神经系统的髓鞘异常。
机译:自闭症相关的SHANK3单倍剂量不足导致人类神经元Ih通道病。
机译:Autism相关的Shank3 HaploUnducks导致人神经元中的IH通道病