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机译:苯的酚类代谢产物诱导的Caspase依赖性细胞毒性对K562细胞伴随着细胞表面唾液酸的减少
School of Biological Science and Medical Engineering, Beihang University, Beijing, 100191, China;
School of Biological Science and Medical Engineering, Beihang University, Beijing, 100191, China;
School of Biological Science and Medical Engineering, Beihang University, Beijing, 100191, China;
School of Biological Science and Medical Engineering, Beihang University, Beijing, 100191, China;
School of Biological Science and Medical Engineering, Beihang University, Beijing, 100191, China;
School of Biological Science and Medical Engineering, Beihang University, Beijing, 100191, China;
School of Biological Science and Medical Engineering, Beihang University, Beijing, 100191, China;
School of Biological Science and Medical Engineering, Beihang University, Beijing, 100191, China;
School of Biological Science and Medical Engineering, Beihang University, Beijing, 100191, China;
apoptosis; benzene metabolites; caspases; reactive oxygen species; sialic acid;
机译:苯的酚类代谢产物抑制K562细胞的红系分化。
机译:DNA-PKcs在苯代谢物的生物学效应中的作用:苯酚对人K562细胞的体外毒性。
机译:唾液酸结合凝集素(酶)诱导Jurkat细胞中胱天蛋白酶依赖性凋亡介导的线粒体扰动
机译:金雀异黄素增强光动力疗法诱导的K562细胞的细胞毒性
机译:苯代谢物诱导的细胞凋亡:NAD(P)H:醌氧化还原酶1(NQO1)C609T多态性对造血细胞对酚类代谢物的敏感性的影响
机译:唾液酸结合凝集素(酶)诱导Jurkat细胞中胱天蛋白酶依赖性凋亡介导的线粒体扰动
机译:唾液酸结合凝集素(leczyme)诱导Jurkat细胞中的依赖依赖性凋亡介导的线粒体扰动