Congenital corneal opacities (CCOs), occurring in approximately 3/100,000 newborns, result from many different disorders, including congenital hereditary endothelial dystrophy (CHED), Peters anomaly (PA), congenital hereditary stromal dystrophy (CHSD), and posterior polymorphous dystrophy (PPMD). CCOs cause visual deprivation during the early months of life that can result in long-term changes to the central nervous system.(1) This may result in profound and uncorrectable loss of vision that can negatively affect a child’s development. Early detection is important to begin appropriate and prompt medical or surgical therapy and minimize amblyopia risk in these children.
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