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Structural interplay of KV7.1 and KCNE1 is essential for normal repolarization and is compromised in short QT syndrome 2 (KV7.1-A287T)

机译：KV7.1和KCNE1的结构相互作用对于正常的复极至关重要并且在短QT综合征2（KV7.1-A287T）中受到损害

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期刊名称 HeartRhythm Case Reports
作者
Ina Rothenberg; Ilaria Piccini; Eva Wrobel; Birgit Stallmeyer; Jovanca Müller; Boris Greber; Nathalie Strutz-Seebohm; Eric Schulze-Bahr; Nicole Schmitt; Guiscard Seebohm;
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作者单位

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年(卷),期 2016(2),6
年度 2016
页码 521–529
总页数 9
原文格式 PDF
正文语种
中图分类
关键词
Long QT syndrome Short QT syndrome Channel Structure Molecular;

机译：长QT综合征;短QT综合征;通道;结构;分子;

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外文文献
专利

1. Amplification of spatial dispersion of repolarization underlies sudden cardiac death associated with catecholaminergic polymorphic VT, long QT, short QT and Brugada syndromes. [J] . Antzelevitch C, Oliva A Journal of Internal Medicine . 2006,第1期

机译：复极化空间分散的放大是与儿茶酚胺能性多形性室速，长QT，短QT和Brugada综合征相关的心源性猝死的基础。
2. Abnormal repolarization dynamics revealed in exercise test in long QT syndrome mutation carriers with normal resting QT interval. [J] . Hekkala AM, Viitasalo M, Vaananen H, Europace: European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology . 2010,第9期

机译：运动测试中发现，在正常QT间隔正常的长QT综合征突变携带者中，异常复极化动力学表现出异常。
3. Abnormal repolarization dynamics revealed in exercise test in long QT syndrome mutation carriers with normal resting QT interval [J] . Anna-Mari Hekkala, Matti Viitasalo, Heikki Väänänen, Europace . 2010,第9期

机译：QT间隔正常的长QT综合征突变携带者在运动测试中发现异常的复极动力学
4. ANALYSIS OF QT INTERVAL PROLONGATION BYSIMULATION OF REPOLARIZATION PROCESS BASED ONKCNQ1 AND KCNE1 EXPRESSION EXPERIMENT [C] . TAKAHIRO YAMAGUCHI, KAICHIRO KAMIYA, TATSUHIKO ARAFUN International Congress on Electrocardiology . 2005

机译：基于KCNQ1和KCNE1表达实验的QT间隔延长QT间隔延长分析
5. A novel transgenic rabbit model with reduced repolarization reserve: long QT syndrome caused by a dominant‐negative mutation of the KCNE1 gene [O] . Péter Major, István Baczkó, László Hiripi, 2016

机译：具有减少的复极储备的新型转基因兔模型：由KCNE1基因的显性负突变引起的长QT综合征
6. Structural interplay of KV7.1 and KCNE1 is essential for normal repolarization and is compromised in short QT syndrome 2 (KV7.1-A287T) [O] . Ina Rothenberg, PhD, Ilaria Piccini, PhD, Eva Wrobel, PhD, 2016

机译：KV7.1和KCNE1的结构相互作用对于正常复极化是必不可少的，并且在短QT综合征2中受到损害（KV7.1-a287T）

Structural interplay of KV7.1 and KCNE1 is essential for normal repolarization and is compromised in short QT syndrome 2 (KV7.1-A287T)

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